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Details
Link-It Detail - PubMed - Glycosylation deficiency at either one of the two glycan attachment sites of cellular prion protein preserves susceptibility to bovine spongiform encephalopathy and scrapie infections.
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PubMed (1)
Glycosylation deficiency at either one of the two glycan attachment sites of cellular prion protein preserves susceptibility to bovine spongiform encephalopathy and scrapie infections. 15448157
Genes (1)

Species:
human : 1
SpeciesGeneGeneIdGene NameEvidence
HumanPRNP5621prion protein
PUBMED, Pubmed Id: 15448157, PUBMED ID: 15448157
XRefs (1)

XRef Types:
pubmed : 1


ToppGene Datasets:
none : 1
ToppGene DatasetsXRef TypeXRef IdXRefValuesSourceTop % Rank
PUBMEDimg 15448157Glycosylation deficiency at either one of the two glycan attachment sites of cellular prion protein preserves susceptibility to bovine spongiform encephalopathy and scrapie infections.0self